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. Nasal carriage can be transiently eradicated by topical administration of the antibiotic mupirocin but this is compromised by the development of resistance [5]. Alternative strategies for reducing nasal carriage are required which will involve a detailed understanding of the molecular basis of interactions between the host and the bacterium that underlie the process.Host factors that determine n
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R (cnc-bZIP) proteins that heterodimerize with small Maf family proteins and activate transcription from anti-oxidant response elements (ARE) in promoters of anti-oxidant defense genes [30]. Nrf2 is normally bound to its suppressor Keap-1 and is inactive in the cytosol, but it dissociates under oxidative stress from Keap-1 and activates the transcription of target genes [23]. Mammalian Bach 1 and